Concurrent RB1 and P53 pathway disruption predisposes to the development of a primitive neuronal component in high-grade gliomas depending on MYC-driven EBF3 transcription
Francesca Pagani(University of Brescia), Pietro Luigi Poliani(University of Brescia), Roberto Ronca(Brescia University), Giovanni Crisafulli(IFOM), Francesca Orzan(University of Milan), Rossella Galli(Vita-Salute San Raffaele University), Daniela Marubbi(Ospedale Policlinico San Martino), Marta Prelli(Candiolo Cancer Institute), Manuela Cominelli(University of Brescia), Francesca De Bacco(Candiolo Cancer Institute), Sara Lago(University of Trento), Magdalena Gryzik(University of Brescia), Maura Poli(University of Brescia), Davide Ceresa(Ospedale Policlinico San Martino), Claudio Isella(Candiolo Cancer Institute), Carla Boccaccio(Department of Medical Sciences), Piera Balzarini(University of Brescia), Paolo Malatesta(Ospedale Policlinico San Martino), Elena Somenza(University of Brescia), Alessio Zippo(University of Trento)
Cited by 6
Related Papers
Induction of MET by Ionizing Radiation and Its Role in Radioresistance and Invasive Growth of Cancer
|JNCI Journal of the National Cancer Institute|2011|382
Selective analysis of cancer-cell intrinsic transcriptional traits defines novel clinically relevant subtypes of colorectal cancer
|Nature Communications|2017|322
Circulating tumor DNA to guide rechallenge with panitumumab in metastatic colorectal cancer: the phase 2 CHRONOS trial
|Nature Medicine|2022|259
A hypomorphic R229Q Rag2 mouse mutant recapitulates human Omenn syndrome
|Journal of Clinical Investigation|2007|111