Expanding the Role of SH2B3: T Cell Large Granular Lymphocytosis (LGL) and Common Variable Immune Deficiency in the Setting of Biallelic SH2B3 Mutations
Tiffany Hangse(Western Michigan University), Eric J. Allenspach(Center for Infectious Disease Research), Lan Beppu(Fred Hutch Cancer Center), Shalini Pullarkat(Fred Hutch Cancer Center), Olga Sala‐Torra(Fred Hutch Cancer Center), Sandhya R. Panch(National Institutes of Health Clinical Center), Cecilia C.S. Yeung(University of Washington), Miranda Galey(University of Washington), Sophie Storz(University of Washington), Aaron Boothby(Fred Hutch Cancer Center), Danny E. Miller(Boston Children's Hospital), Maite de la Morena(University of Washington)
Cited by 2
Related Papers
MHC class II–dependent basophil–CD4+ T cell interactions promote TH2 cytokine–dependent immunity
|Nature Immunology|2009|590
Notch Signaling in Cancer
|Cancer Biology & Therapy|2002|341
Jakinibs for the treatment of immune dysregulation in patients with gain-of-function signal transducer and activator of transcription 1 (STAT1) or STAT3 mutations
|Journal of Allergy and Clinical Immunology|2018|261
ERM-Dependent Movement of CD43 Defines a Novel Protein Complex Distal to the Immunological Synapse
|Immunity|2001|252
Targeted long-read sequencing identifies missing disease-causing variation
|The American Journal of Human Genetics|2021|239