X-linked deletion of Crossfirre, Firre, and Dxz4 in vivo uncovers diverse phenotypes and combinatorial effects on autosomes
Tim P. Hasenbein(German Centre for Cardiovascular Research), Daniel Andergassen(German Centre for Cardiovascular Research), Birgit Rathkolb(Helmholtz Zentrum München), Patricia da Silva‐Buttkus(Helmholtz Munich), John L. Rinn(University of Colorado Boulder), Nathalia Romanelli Vicente Dragano(Helmholtz Munich), Markus Kraiger(Helmholtz Zentrum München), Wolfgang Wurst(German Center for Neurodegenerative Diseases), Juan Antonio Aguilar‐Pimentel(Helmholtz Zentrum München), Alexander Meissner(Max Planck Institute for Molecular Genetics), Zachary D. Smith(St. Jude Children's Research Hospital), Sabine M. Hölter(Helmholtz Zentrum München), Oana V. Amarie(Helmholtz Munich), Valérie Gailus‐Durner, Manuela A. Östereicher(Helmholtz Munich), Adrián Sanz‐Moreno(Helmholtz Munich), Chiara Gerhardinger(Massachusetts Eye and Ear Infirmary), Stefan Engelhardt(Heidelberg University), Lore Becker(Ludwig-Maximilians-Universität München), Nadine Spielmann(Helmholtz Munich), Lillian Garrett(Helmholtz Zentrum München), Julia Calzada-Wack(Helmholtz Munich), Sarah Hoelzl(German Centre for Cardiovascular Research), Martin Hrabě de Angelis(Helmholtz Zentrum München), Marion O. C. Gonner(German Centre for Cardiovascular Research), Helmut Fuchs(Helmholtz Zentrum München)
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