Mutagenic DNA Lesions in Normal Hematopoietic Stem Cells Persist for Years Causing Distinct Patterns of Mutation
Michael Spencer Chapman(Wellcome Sanger Institute), Peter J. Campbell(Wellcome Sanger Institute), C. Matthias Wilk(Icahn School of Medicine at Mount Sinai), Kenichi Yoshida(Wellcome Sanger Institute), Iñigo Martincorena(Wellcome Sanger Institute), George S. Vassiliou(Wellcome/MRC Cambridge Stem Cell Institute), Krishnaa T. Mahbubani(University of Cambridge), Anna Maria Ranzoni(Wellcome/MRC Cambridge Stem Cell Institute), Emily Mitchell(Wellcome/MRC Cambridge Stem Cell Institute), Ana Cvejic(University of Copenhagen), Philip S. Robinson(Wellcome Sanger Institute), Kourosh Saeb‐Parsy(University of Cambridge), Kate H.C. Gowers(University College London), Margarete A. Fabre(University of Cambridge), Michael R. Stratton(Wellcome Sanger Institute), Tim Coorens(Broad Institute), Steffen Boettcher(University Hospital of Zurich), Sam M. Janes(University College London), Nicholas Williams(Wellcome Sanger Institute), Matt Hoare(Cancer Research UK Cambridge Center), Elisa Laurenti(Wellcome/MRC Cambridge Stem Cell Institute), Jyoti Nangalia(Wellcome Sanger Institute), Lori D. Kregar(Wellcome Sanger Institute), Anthony R Green(Wellcome/MRC Cambridge Stem Cell Institute), Stanley Ng(University of Toronto), Markus G. Manz(Ospedale San Giovanni Bellinzona)
Cited by 1
Related Papers
COSMIC: the Catalogue Of Somatic Mutations In Cancer
|Nucleic Acids Research|2018|5.2k
Genomic Classification and Prognosis in Acute Myeloid Leukemia
|New England Journal of Medicine|2016|4.4k
The cancer genome
|Nature|2009|3.5k
Acquired mutation of the tyrosine kinase JAK2 in human myeloproliferative disorders
|The Lancet|2005|3.4k