Enhancing transcription–replication conflict targets ecDNA-positive cancers

Jun Tang(Hubei University of Medicine), Howard Y. Chang(Stanford University), Sudhir Chowdhry(Boundless Bio (United States)), Anthony B. Pinkerton(Boundless Bio (United States)), Vineet Bafna(University of California San Diego), Christian A. Hassig(Boundless Bio (United States)), Christina Curtis(Chan Zuckerberg Initiative (United States)), Ellis J. Curtis(University of California San Diego), Paul S. Mischel(University of California, Los Angeles), Guiping Wang(Soochow University), Ivy Tsz-Lo Wong(Stanford University), Philip Hanoian(Pennsylvania State University), Auzon Steffy(Genomics Institute of the Novartis Research Foundation), Yanding Zhao(Stanford University), Stephen J. Benkovic(Pennsylvania State University), Nicolas Altemose(Stanford University), Snezana Milutinovic(Sierra Wireless (Canada)), Jens Luebeck(University of Washington Tacoma), William J. Greenleaf(Stanford University), Salvador Garcia Mojica(Boundless Bio (United States)), Natasha E. Weiser(Stanford University), Shailaja Kasibhatla(Genomics Institute of the Novartis Research Foundation), Joshua Plum(Boundless Bio (United States)), Rui Li(Sichuan University), Shu Zhang(Stanford University), Yanbo Wang(Stanford University), S. Todd Meyer(Boundless Bio (United States)), Georgi K. Marinov(Stanford University), Ryan J. Hansen(Boundless Bio (United States)), Edison Tse(Boundless Bio (United States))
Nature
November 6, 2024
Cited by 88


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