Abstract 63: Acetylation of Mitochondrial Cyclophilin D Increases vascular Oxidative Stress, Induces Glycolitic Switch, Promotes Endothelial Dysfunction and Hypertension
Sergey Dikalov(Vanderbilt University Medical Center), Anna Dikalova(Vanderbilt University Medical Center), Venkataraman Amarnath(Vanderbilt University), Mingfang Ao(Vanderbilt University Medical Center), Frederic T. Billings, Daniel J. Fehrenbach(Indiana University – Purdue University Indianapolis), Michael N. Sack(National Institutes of Health), Louise Lantier(Vanderbilt University), Alexander Panov(Mercer University), Vladimir Mayorov(Mercer University), Marcos G. Lopez(Vanderbilt University Medical Center)
Cited by 2
Related Papers
Mitochondrial reactive oxygen species promote production of proinflammatory cytokines and are elevated in TNFR1-associated periodic syndrome (TRAPS)
|The Journal of Experimental Medicine|2011|854
Fatty Acid Oxidation Enzyme Gene Expression Is Downregulated in the Failing Heart
|Circulation|1996|681
Nitrite augments tolerance to ischemia/reperfusion injury via the modulation of mitochondrial electron transfer
|The Journal of Experimental Medicine|2007|561
DC isoketal-modified proteins activate T cells and promote hypertension
|Journal of Clinical Investigation|2014|533