Total loss of <i>VHL</i> gene function impairs neuroendocrine cancer cell fitness due to excessive HIF2α activity
Muhannad Abu‐Remaileh(Hebrew University of Jerusalem), William G. Kaelin(Dana-Farber Cancer Institute)
Cited by 9
Related Papers
An intact NEDD8 pathway is required for Cullin‐dependent ubiquitylation in mammalian cells
|EMBO Reports|2002|179
The KDM5A/RBP2 histone demethylase represses NOTCH signaling to sustain neuroendocrine differentiation and promote small cell lung cancer tumorigenesis
|Genes & Development|2019|130
Phosphorylation of ETS1 by Src Family Kinases Prevents Its Recognition by the COP1 Tumor Suppressor
|Cancer Cell|2014|89
Tumor suppressor WWOX regulates glucose metabolism via HIF1α modulation
|Cell Death and Differentiation|2014|85