Subset-specific mitochondrial stress and DNA damage shape T cell responses to fever and inflammation
Darren R. Heintzman(Vanderbilt University Medical Center), Jeffrey C. Rathmell(Vanderbilt University Medical Center)
Cited by 29
Related Papers
Cutting Edge: Distinct Glycolytic and Lipid Oxidative Metabolic Programs Are Essential for Effector and Regulatory CD4+ T Cell Subsets
|The Journal of Immunology|2011|2k
Glucose Uptake Is Limiting in T Cell Activation and Requires CD28-Mediated Akt-Dependent and Independent Pathways
|The Journal of Immunology|2008|836
Mitochondrial dysregulation and glycolytic insufficiency functionally impair CD8 T cells infiltrating human renal cell carcinoma
|JCI Insight|2017|380
Immunometabolism: From basic mechanisms to translation
|Immunological Reviews|2020|372
Activated Akt promotes increased resting T cell size, CD28‐independent T cell growth, and development of autoimmunity and lymphoma
|European Journal of Immunology|2003|194