Knockout of Dectin-1 does not modify disease onset or progression in a MATR3 S85C knock-in mouse model of ALS
Justin You(Hospital for Sick Children), Jeehye Park(University of Toronto)
Cited by 2
Related Papers
Knockdown of genes involved in axonal transport enhances the toxicity of human neuromuscular disease‐linked MATR3 mutations in <i>Drosophila</i>
|FEBS Letters|2020|11
<scp>MATR3</scp> pathogenic variants differentially impair its cryptic splicing repression function
|FEBS Letters|2024|11
Differential effects of MATR3 variants on its cryptic splicing repression function
|bioRxiv (Cold Spring Harbor Laboratory)|2023|0
Knockout of Dectin-1 does not modify disease onset or progression in a MATR3 S85C knock-in mouse model of ALS
|bioRxiv (Cold Spring Harbor Laboratory)|2024|0