Cellular prion protein acts as mediator of amyloid beta uptake by caveolin‐1 causing cellular dysfunctions in vitro and in vivo
Angela da Silva Correia(German Center for Neurodegenerative Diseases), Inga Zerr(Universitätsmedizin Göttingen), Tiago F. Outeiro(German Center for Neurodegenerative Diseases), André Fischer(German Center for Neurodegenerative Diseases), Amandeep Singh Arora(Brown Foundation), Franco L. Simonetti(Fundación Instituto Leloir), Gesine Saher(Max Planck Institute of Experimental Medicine), Anna‐Lisa Fischer(German Center for Neurodegenerative Diseases), Roberto Goya‐Maldonado(Social Neuroscience Lab), Susana Da Silva Correia(German Center for Neurodegenerative Diseases), Matthias Schmitz(University of Göttingen)
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