The pathogenic T42A mutation in SHP2 rewires the interaction specificity of its N-terminal regulatory domainAnne E. van Vlimmeren(Columbia University), Neel H. Shah(QB3)Proceedings of the National Academy of SciencesJuly 16, 202410.1073/pnas.2407159121Cited by 18SaveCiteExport RISWatch citationsRelated PapersAn electrostatic selection mechanism controls sequential kinase signaling downstream of the T cell receptor|eLife|2016|102High-throughput profiling of sequence recognition by tyrosine kinases and SH2 domains using bacterial peptide display|eLife|2023|25