Mitochondrial dysfunction and metabolic reprogramming induce macrophage pro-inflammatory phenotype switch and atherosclerosis progression in agingAleksandr E. Vendrov(University of Michigan), Nageswara R. Madamanchi(Michigan Medicine)Frontiers in ImmunologyJune 21, 202410.3389/fimmu.2024.1410832Cited by 41SaveCiteExport RISWatch citationsRelated PapersMitochondrial oxidative stress contributes to diastolic dysfunction through impaired mitochondrial dynamics|Redox Biology|2022|99Cardiomyocyte NOX4 regulates resident macrophage-mediated inflammation and diastolic dysfunction in stress cardiomyopathy|Redox Biology|2023|52Leukocyte Antigen-related Protein Tyrosine Phosphatase Negatively Regulates Hydrogen Peroxide-induced Vascular Smooth Muscle Cell Apoptosis|UNC Libraries|2020|2