The Alzheimer’s disease–linked protease BACE2 cleaves VEGFR3 and modulates its signaling
Andree Schmidt(TUM Klinikum), Stefan F. Lichtenthaler(German Center for Neurodegenerative Diseases)
Cited by 14
Related Papers
ADAM10 is the physiologically relevant, constitutive α‐secretase of the amyloid precursor protein in primary neurons
|The EMBO Journal|2010|597
Secretome protein enrichment identifies physiological BACE1 protease substrates in neurons
|The EMBO Journal|2012|315
Regulated intramembrane proteolysis - lessons from amyloid precursor protein processing
|Journal of Neurochemistry|2011|233
The case for low-level BACE1 inhibition for the prevention of Alzheimer disease
|Nature Reviews Neurology|2021|136