Abstract PR010: <i>CDK12</i> loss promotes prostate cancer development while exposing vulnerabilities to paralog-based synthetic lethality
Jean C. Tien(University of Michigan), Arul M. Chinnaiyan(Howard Hughes Medical Institute), Yuping Zhang(University of Michigan), Jianzhang Yang(Jinan University), Xiao Ming Wang(Chinese Academy of Agricultural Sciences), Rachel Brough(Institute of Cancer Research), Jonathan Chou(University of California, San Francisco), Sanjana Eyunni(University of Michigan), Xuhong Cao(Howard Hughes Medical Institute), James Neiswender(Institute of Cancer Research), Marcin Cieślik(University of Michigan), Rahul Mannan(University of Michigan), Estefanía Labanca(The University of Texas MD Anderson Cancer Center), Yi Bao(Michigan Center for Translational Pathology), Yu Chang(University of Michigan), Stephen J. Pettitt(Breast Cancer Now), Christopher J. Lord(Institute of Cancer Research), Yuzhuo Wang(BC Cancer Agency), Xiaoju Wang(Zhejiang Academy of Medical Sciences), Yunhui Cheng(University of Michigan), Caleb Cheng(University of Michigan), Ke Ding(Chinese Academy of Sciences), Abbey Todd(University of Michigan), Palak Shah(University of Chicago)
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