Neutrophils disrupt B-1a cell homeostasis by targeting Siglec-G to exacerbate sepsis
Cited by 15
Related Papers
NAD+ consumption by PARP1 in response to DNA damage triggers metabolic shift critical for damaged cell survival
|Molecular Biology of the Cell|2019|222
Inhibition of aerobic glycolysis alleviates sepsis‑induced acute kidney injury by promoting lactate/Sirtuin 3/AMPK‑regulated autophagy
|International Journal of Molecular Medicine|2021|153
STAT3–BDNF–TrkB signalling promotes alveolar epithelial regeneration after lung injury
|Nature Cell Biology|2020|143
Neutralization of osteopontin attenuates neutrophil migration in sepsis-induced acute lung injury
|Critical Care|2015|129
Extracellular CIRP as an endogenous TREM-1 ligand to fuel inflammation in sepsis
|JCI Insight|2020|128