Abstract PO2-25-01: Tumor Derived Chitinase-3 like 1 Induces Neutrophil Extracellular Traps that Promote T cell Exclusion and Resistance to Immunotherapies in Breast Cancer
Tarek Taifour(McGill University), William A. Müller(Northwestern University), Sherif Samer Attalla(McGill University), Jack A. Elias(Yale University), Suchitra Kamle(Brown University), Hellen Kuasne(McGill University Health Centre), Vasilios Papavasiliou(McGill University), Emilie Solymoss(McGill University Health Centre), Peter M. Siegel(L'Oréal (France)), Morag Park(McGill University Health Centre), Chun Geun Lee(Brown University), Hailey Dall-Proud(McGill University), Yu Gu(McGill University Health Centre), Virginie Sanguin‐Gendreau(McGill University), Dongmei Zuo(McGill University), Tung Bui(McGill University)
Cited by 1
Related Papers
Pulmonary expression of interleukin-13 causes inflammation, mucus hypersecretion, subepithelial fibrosis, physiologic abnormalities, and eotaxin production
|Journal of Clinical Investigation|1999|1.7k
Endocytosis and the recycling of plasma membrane.
|The Journal of Cell Biology|1983|1.5k
PECAM-1 is required for transendothelial migration of leukocytes.
|The Journal of Experimental Medicine|1993|1.1k
PECAM-1 (CD31) Cloning and Relation to Adhesion Molecules of the Immunoglobulin Gene Superfamily
|Science|1990|954
Interleukin-13 Induces Tissue Fibrosis by Selectively Stimulating and Activating Transforming Growth Factor β1
|The Journal of Experimental Medicine|2001|926