Phosphoproteomic Analysis Identified Mutual Phosphorylation of FAK and Src as a Mechanism of Osimertinib Resistance in EGFR-Mutant Lung CancerTakehiro Tozuka(Nippon Medical School), Masahiro Seike(Nippon Medical School Hospital)JTO Clinical and Research ReportsMarch 22, 202410.1016/j.jtocrr.2024.100668Cited by 7SaveCiteExport RISWatch citationsRelated PapersAXL and GAS6 co-expression in lung adenocarcinoma as a prognostic classifier|Oncology Reports|2017|19Afatinib plus PEM and CBDCA overcome osimertinib resistance in <i>EGFR</i>‐mutated NSCLC with high thrombospondin‐1 expression|Cancer Science|2024|7Induction of resistance to neurotrophic tropomyosin‐receptor kinase inhibitors by <scp>HMGCS2</scp> via a mevalonate pathway|Cancer Medicine|2024|4