Xenografted human microglia display diverse transcriptomic states in response to Alzheimer’s disease-related amyloid-β pathology
Renzo Mancuso(University of Antwerp), Bart De Strooper(Allen Institute for Brain Science), Sriram Balusu(VIB-KU Leuven Center for Brain & Disease Research), Lutgarde Serneels(VIB-KU Leuven Center for Brain & Disease Research), Johanna Van Den Daele(Leuven Institute for Fertility and Embryology), Catherine M. Verfaillie(University of Minnesota), Leen Wolfs(VIB-KU Leuven Center for Brain & Disease Research), Mark Fiers(UK Dementia Research Institute), Baukje Bijnens(University of Antwerp), Suresh Poovathingal(VIB-KU Leuven Center for Brain & Disease Research), Emma Davis(UK Dementia Research Institute), Anna Martínez‐Muriana(Vlaams Instituut voor Biotechnologie), Pranav Preman(VIB-KU Leuven Center for Brain & Disease Research), Jessie Premereur(University of Antwerp), Ivana Geric(Vlaams Instituut voor Biotechnologie), Paula Polanco(University of Antwerp), Nicola Fattorelli(Vlaams Instituut voor Biotechnologie)
Cited by 123
Related Papers
Donor leukocyte infusions in 140 patients with relapsed malignancy after allogeneic bone marrow transplantation.
|Journal of Clinical Oncology|1997|1.3k
Destabilization of β-catenin by mutations in presenilin-1 potentiates neuronal apoptosis
|Nature|1998|517
Alteration of the microRNA network during the progression of Alzheimer's disease
|EMBO Molecular Medicine|2013|476
Presenilins and -Secretase: Structure, Function, and Role in Alzheimer Disease
|Cold Spring Harbor Perspectives in Medicine|2011|461