Abstract 133: GMDS is a key driver of <i>MYCN</i>-amplified neuroblastoma core fucosylation and tumorigenesis
Beibei Zhu(University of Kentucky), Eric J. Rellinger(University of Kentucky), Oscar Lopez‐Nunez(Cincinnati Children's Hospital Medical Center), Michael D. Buoncristiani(University of Kentucky), Lindsay Bryant(University of Kentucky), Cameron J. Shedlock(University of Florida), Ramon C. Sun(University of Florida), Derek B. Allison(University of Kentucky), Juan P. Gurría(Cincinnati Children's Hospital Medical Center), Roberto A. Ribas(University of Florida), B. Mark Evers(University of Kentucky), Michelle G. Pitts(University of Kentucky), Nathan Shelman(Indiana University School of Medicine), Shannon Keohane(University of Florida), Matt Gentry(University of Florida), Chi Wang(Markey Cancer Center), Jinpeng Liu(University of Kentucky)
Cited by 0
Related Papers
mTORC1 and mTORC2 Regulate EMT, Motility, and Metastasis of Colorectal Cancer via RhoA and Rac1 Signaling Pathways
|Cancer Research|2011|539
Ketogenesis contributes to intestinal cell differentiation
|Cell Death and Differentiation|2016|129
Brain glycogen serves as a critical glucosamine cache required for protein glycosylation
|Cell Metabolism|2021|122
p300/CBP inhibition enhances the efficacy of programmed death-ligand 1 blockade treatment in prostate cancer
|Oncogene|2020|114
Deptor Is a Novel Target of Wnt/β-Catenin/c-Myc and Contributes to Colorectal Cancer Cell Growth
|Cancer Research|2018|104