Data from Ecdysoneless Overexpression Drives Mammary Tumorigenesis through Upregulation of C-MYC and Glucose Metabolism
Bhopal Mohapatra, Vimla Band(Nebraska Medical Center), Emad A. Rakha(Nottingham University Hospitals NHS Trust), Sameer Mirza(Nebraska Medical Center), Chittibabu Guda, Siddesh Southekal, Kay‐Uwe Wagner(Nebraska Medical Center), Robert D. Cardiff(University of California, Davis), Surendra K. Shukla(University of Oklahoma Health Sciences Center), Aditya Bele, Channabasavaiah B. Gurumurthy(Nebraska Medical Center), Mansour Alsaleem(University of Nottingham), Irfana Saleem, Matthew D. Storck, Aniruddha Sarkar, Sai Sundeep Kollala, Fang Qiu, Subodh M. Lele, Hamid Band(Nebraska Medical Center), Mohsin Raza, Pankaj Kumar Singh(Lovely Professional University)
Cited by 0
Related Papers
Easi-CRISPR: a robust method for one-step generation of mice carrying conditional and insertion alleles using long ssDNA donors and CRISPR ribonucleoproteins
|Genome biology|2017|569
Protein kinase CK2 in mammary gland tumorigenesis
|Oncogene|2001|296
Activation of Akt (Protein Kinase B) in Mammary Epithelium Provides a Critical Cell Survival Signal Required for Tumor Progression
|Molecular and Cellular Biology|2001|279
Metabolic reprogramming induced by ketone bodies diminishes pancreatic cancer cachexia
|Cancer & Metabolism|2014|253
i-GONAD: a robust method for in situ germline genome engineering using CRISPR nucleases
|Genome biology|2018|235