A syngeneic spontaneous zebrafish model of tp53-deficient, EGFRviii, and PI3KCAH1047R-driven glioblastoma reveals inhibitory roles for inflammation during tumor initiation and relapse in vivo
A. H. Weiss(Oregon Health & Science University), Madeline N. Hayes(University of Toronto)
Cited by 0
Related Papers
A syngeneic spontaneous zebrafish model of <i>tp53</i> -deficient, EGFR <sup>vIII</sup> , and PI3KCA <sup>H1047R</sup> -driven glioblastoma reveals inhibitory roles for inflammation during tumor initiation and relapse <i>in vivo</i>
|bioRxiv (Cold Spring Harbor Laboratory)|2023|0
DNA Damage Response Deficiency Enhances Neuroblastoma Progression and Sensitivity to Combination PARP and ATR Inhibition
|bioRxiv (Cold Spring Harbor Laboratory)|2024|0