Metabolic reprograming and increased inflammation by cadmium exposure following early-life respiratory syncytial virus infection: the involvement of protein S-palmitoylation
Zachery Jarrell(Emory University), Young‐Mi Go(University of Atlanta)
Cited by 15
Related Papers
H2O2-dependent Activation of GCLC-ARE4 Reporter Occurs by Mitogen-activated Protein Kinase Pathways without Oxidation of Cellular Glutathione or Thioredoxin-1
|Journal of Biological Chemistry|2004|98
Reactive Aldehyde Modification of Thioredoxin-1 Activates Early Steps of Inflammation and Cell Adhesion
|American Journal Of Pathology|2007|89
Disturbed Flow Enhances Inflammatory Signaling and Atherogenesis by Increasing Thioredoxin-1 Level in Endothelial Cell Nuclei
|PLoS ONE|2014|36
Increased Inflammatory Signaling and Lethality of Influenza H1N1 by Nuclear Thioredoxin-1
|PLoS ONE|2011|34
H2S Inhibits Oscillatory Shear Stress-Induced Monocyte Binding to Endothelial Cells Via Nitric Oxide Production
|Molecules and Cells|2012|18