Gαs is dispensable for β-arrestin coupling but dictates GRK selectivity and is predominant for gene expression regulation by β2-adrenergic receptor
Valeria Burghi(University of California San Diego), Justine S. Paradis(University of California San Diego), Adam Officer(University of California San Diego), Sendi Rafael Adame-Garcia(University of California San Diego), Xingyu Wu(University of California San Diego), Edda S. F. Matthees(Jena University Hospital), Benjamin Barsi‐Rhyne(University of California, San Francisco), Dana J. Ramms(University of California San Diego), Lauren Clubb(University of California San Diego), Monica Acosta(University of California San Diego), Pablo Tamayo(University of California San Diego), Michel Bouvier(Institute for Research in Immunology and Cancer), Asuka Inoue(Tohoku University), Mark von Zastrow(University of California, San Francisco), Carsten Hoffmann(Jena University Hospital), J. Silvio Gutkind(University of California San Diego)
Cited by 15Open Access
Abstract
Gs is dispensable for -arrestin coupling but dictates GRK selectivity and is predominant for gene expression regulation by 2-adrenergic receptor
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