Genetic inactivation of zinc transporter SLC39A5 improves liver function and hyperglycemia in obesogenic settings
Shek Man Chim(Regeneron (United States)), Harikiran Nistala(Icahn School of Medicine at Mount Sinai), Alan R. Shuldiner(Regeneron (United States)), John Dronzek(Regeneron (United States)), Jonas Bovijn(Regeneron (United States)), Luca Lotta(Regeneron (United States)), Cristopher V. Van Hout(Regeneron (United States)), Adam E. Locke(James S. McDonnell Foundation), Anthony Marcketta(Regeneron (United States)), Susannah Brydges(Regeneron (United States)), Kristen E. Howell(Texas A&M University), Tanima De(Regeneron (United States)), Vinayagam Arunachalam(Regeneron (United States)), Katia Karalis(Regeneron (United States)), Manuel A. R. Ferreira(Broad Institute), David J. Carey(Geisinger Medical Center), Olle Melander(Lund University), Michelle G. LeBlanc(Toronto General Hospital), Weizhen Wu(Third Affiliated Hospital of Guangzhou Medical University), Bin Ye(Regeneron (United States)), Alexander Li(Regeneron (United States)), Aris N. Economides(Regeneron (United States)), Lyndon J. Mitnaul(Regeneron (United States)), Niek Verweij(Regeneron (United States))
Cited by 1
Related Papers
Genetic Risk, Adherence to a Healthy Lifestyle, and Coronary Disease
|New England Journal of Medicine|2016|1.5k
Collaborative genome-wide association analysis supports a role for ANK3 and CACNA1C in bipolar disorder
|Nature Genetics|2008|1.3k
Identification of 64 Novel Genetic Loci Provides an Expanded View on the Genetic Architecture of Coronary Artery Disease
|Circulation Research|2017|1.2k
Genome-wide association and Mendelian randomisation analysis provide insights into the pathogenesis of heart failure
|Nature Communications|2020|924