Loss-of-function mutations in Dnmt3a and Tet2 lead to accelerated atherosclerosis and concordant macrophage phenotypes
Philipp J. Rauch(Broad Institute), Siddhartha Jaiswal(Otsuka (Japan)), Amélie Vromman(Brigham and Women's Hospital), Kameron B. Rodrigues(Stanford University), Drew N. Cohen(Dana-Farber Cancer Institute), Manu Gopakumar(Stanford University), Alexander J. Silver(Brigham and Women's Hospital), Herra Ahmad(Stanford University), Peter Libby(Brigham and Women's Hospital), Sean C. Bendall(Stanford University), Zumana Khair(Stanford University), Thiago Rentz, Jayakrishnan Gopakumar(Stanford University), Erin McCaffrey(Stanford University), Amy E. Lin(Brigham and Women's Hospital), Eugenia Shvartz(Brigham and Women's Hospital), Noah F. Greenwald(Broad Institute), Daniel Nachun(Stanford University), Tetsushi Nakao(Broad Institute), Benjamin L. Ebert(Otsuka (Japan)), Maia Fefer(Brigham and Women's Hospital), Eti Sinha(Stanford University), Marc Bossé(Stanford University), Galina K. Sukhova(Brigham and Women's Hospital), Nora Vivanco Gonzalez(Stanford University), Marie McConkey(Dana-Farber Cancer Institute), Michael Angelo(Stanford Medicine)
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