Endothelial activation and fibrotic changes are impeded by laminar flow-induced CHK1-SENP2 activity through mechanisms distinct from endothelial-to-mesenchymal cell transition
Minh Thi Nguyen(Houston Methodist), Nhat-Tu Le(Houston Methodist), Anita Deswal(The University of Texas MD Anderson Cancer Center), Nicolas L. Palaskas(The University of Texas MD Anderson Cancer Center), Elizabeth A. Olmsted‐Davis(William & Mary), John P. Cooke(Houston Methodist), Loka Reddy Velatooru(Houston Methodist), Khanh Chau(Houston Methodist), Jun‐ichi Abe(The University of Texas MD Anderson Cancer Center), Venkata Subrahman K Samanthapudi(The University of Texas MD Anderson Cancer Center), Masaki Imanishi(Tokushima University Hospital), Elena McBeath(The University of Texas MD Anderson Cancer Center), Steven H. Lin(The University of Texas MD Anderson Cancer Center), Robert Dantzer(The University of Texas MD Anderson Cancer Center), Mae K. Borchrdt(Houston Methodist), Sivareddy Kotla(University of Tennessee Health Science Center), Estefani Berrios Turcios(Houston Methodist), Guangyu Wang(Tongji University), Priyanka Banerjee(Adamas University), Kyung Ae Ko(The University of Texas MD Anderson Cancer Center), Keigi Fujiwara(Norwegian Womens Public Health Association), Shengyu Li(Houston Methodist), Young Jin Gi(The University of Texas MD Anderson Cancer Center), Ling-Ling Lee(The University of Texas MD Anderson Cancer Center), Rei Abe(Houston Methodist)
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