MAVS Positively Regulates Mitochondrial Integrity and Metabolic Fitness in B Cells
Hongsheng Wang(Donghua University), Silvia Bolland(National Institute of Allergy and Infectious Diseases), Juan Wu(Chinese Academy of Medical Sciences & Peking Union Medical College), Javier Traba(National Institutes of Health), Bethany Scott(National Institutes of Health), Chen‐Feng Qi(National Institutes of Health), Komudi Singh(National Institutes of Health), Michael N. Sack(National Institutes of Health), Wenxiang Sun(Scripps Research Institute), Hemanta K. Kole(National Institutes of Health), Laura Amo
Cited by 4
Related Papers
ERIS, an endoplasmic reticulum IFN stimulator, activates innate immune signaling through dimerization
|Proceedings of the National Academy of Sciences|2009|864
Mitochondrial reactive oxygen species promote production of proinflammatory cytokines and are elevated in TNFR1-associated periodic syndrome (TRAPS)
|The Journal of Experimental Medicine|2011|854
Fatty Acid Oxidation Enzyme Gene Expression Is Downregulated in the Failing Heart
|Circulation|1996|681
Nitrite augments tolerance to ischemia/reperfusion injury via the modulation of mitochondrial electron transfer
|The Journal of Experimental Medicine|2007|561