A Synthetic Poly(A) Tail Targeting Extracellular CIRP Inhibits Sepsis
Atsushi Murao(Feinstein Institute for Medical Research), Ping Wang(Guangzhou Women and Children Medical Center)
Cited by 13
Related Papers
NAD+ consumption by PARP1 in response to DNA damage triggers metabolic shift critical for damaged cell survival
|Molecular Biology of the Cell|2019|222
Neutralization of osteopontin attenuates neutrophil migration in sepsis-induced acute lung injury
|Critical Care|2015|129
Extracellular CIRP as an endogenous TREM-1 ligand to fuel inflammation in sepsis
|JCI Insight|2020|128
The Fbw7/Human CDC4 Tumor Suppressor Targets Proproliferative Factor KLF5 for Ubiquitination and Degradation through Multiple Phosphodegron Motifs
|Journal of Biological Chemistry|2010|113