A pro-inflammatory stem cell niche drives myelofibrosis through a targetable galectin 1 axis
Rong Li(Children's of Alabama), Bethan Psaila(University of Oxford), Hosuk Ryou(John Radcliffe Hospital), Anandi Krishnan(Pennsylvania State University), Supat Thongjuea(University of Oxford), Vijay Rathinam(UConn Health), Pengwei Dong(Fudan University), M. Colombo(Human Technopole), Zoë C. Wong(National Institutes of Health), Daniel Royston(John Radcliffe Hospital), Gabriel A. Rabinovich(Universidad de Buenos Aires), Charlotte Brierley(University of Oxford), Sally‐Ann Clark(National Institute for Health and Care Research), Adam J. Mead(National Institute for Health and Care Research), Aude-Anaïs Olijnik(National Institute for Health and Care Research), Natalie J. Jooss(MRC Weatherall Institute of Molecular Medicine), Yiran Meng(University of Oxford), Matthew Clarke(Institute of Cancer Research), Ruggiero Norfo(University of Oxford), Juan M. Pérez Sáez(Universidad de Buenos Aires), Abdullah O. Khan(National Institute for Health and Care Research), Korsuk Sirinukunwattana(University of Warwick), Zemin Ren(Jiangnan University), Lauren C. Murphy(MRC Weatherall Institute of Molecular Medicine), Guanlin Wang(Fudan University), Christina Simoglou Karali(MRC Weatherall Institute of Molecular Medicine), Antonio Rodriguez-Romera(National Health Service), Joana Carrelha(University of Oxford), Qian Cheng(North Sichuan Medical University), Wenjiang Zhou(Fudan University), Jennifer O’Sullivan(MRC Weatherall Institute of Molecular Medicine), Sean Wen(Wellcome/MRC Cambridge Stem Cell Institute), Nikolaos Sousos(John Radcliffe Hospital)
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