Mitochondria–Endoplasmic Reticulum Contact Sites Dynamics and Calcium Homeostasis Are Differentially Disrupted in <scp><i>PINK1</i>‐PD</scp> or <scp><i>PRKN</i>‐PD</scp> Neurons
Dajana Großmann(University of Luxembourg), Andreas Hermann(German Center for Neurodegenerative Diseases)
Cited by 29
Related Papers
Impaired DNA damage response signaling by FUS-NLS mutations leads to neurodegeneration and FUS aggregate formation
|Nature Communications|2018|288
Hot-spot KIF5A mutations cause familial ALS
|Brain|2017|231
Small molecule modulation of a redox-sensitive stress granule protein dissolves stress granules with beneficial outcomes for familial amyotrophic lateral sclerosis models
|bioRxiv (Cold Spring Harbor Laboratory)|2019|91
Mutations in <i>RHOT1</i> Disrupt Endoplasmic Reticulum–Mitochondria Contact Sites Interfering with Calcium Homeostasis and Mitochondrial Dynamics in Parkinson's Disease
|Antioxidants and Redox Signaling|2019|84
Small-molecule dissolution of stress granules by redox modulation benefits ALS models
|Nature Chemical Biology|2025|37