An uncanonical CDK6 activity inhibits cilia function by suppressing axoneme polyglutamylation
Kai He(Mayo Clinic), Jinghua Hu(Mayo Clinic)
Cited by 0
Related Papers
Candidate exome capture identifies mutation of SDCCAG8 as the cause of a retinal-renal ciliopathy
|Nature Genetics|2010|318
Transition fibre protein FBF1 is required for the ciliary entry of assembled intraflagellar transport complexes
|Nature Communications|2013|124
Axoneme polyglutamylation regulated by Joubert syndrome protein ARL13B controls ciliary targeting of signaling molecules
|Nature Communications|2018|100
A stress-induced cilium-to-PML-NB route drives senescence initiation
|Nature Communications|2023|14
Non-canonical CDK6 activity promotes cilia disassembly by suppressing axoneme polyglutamylation
|The Journal of Cell Biology|2024|12