Cholecystokinin Signaling can Rescue Cognition and Synaptic Plasticity in the APP/PS1 Mouse Model of Alzheimer’s Disease

Zijuan Zhang(Chinese Academy of Inspection and Quarantine), Christian Hölscher(Shanxi Medical University), Z. Zhang(First Affiliated Hospital of Henan University), Shijie Wang(First Affiliated Hospital of Henan University), Ye Yuan(First Affiliated Hospital of Henan University), Hao Li(First Affiliated Hospital of Henan University), Jinlian Ma(First Affiliated Hospital of Henan University), Li Hao(First Affiliated Hospital of Henan University), Shijie Wang(First Affiliated Hospital of Henan University), Zhonghua Li(First Affiliated Hospital of Henan University), Ye Yuan(First Affiliated Hospital of Henan University), Zhenqiang Zhang(First Affiliated Hospital of Henan University)
Molecular Neurobiology
May 29, 2023
10.1007/s12035-023-03388-7
Cited by 22

Related Papers

Aβ42‐driven cerebral amyloidosis in transgenic mice reveals early and robust pathology
|EMBO Reports|2006|1k
Untargeted Metabolomic Analysis of Human Plasma Indicates Differentially Affected Polyamine and L-Arginine Metabolism in Mild Cognitive Impairment Subjects Converting to Alzheimer’s Disease
|PLoS ONE|2015|192
Metabolomic Profiling of Bile Acids in Clinical and Experimental Samples of Alzheimer’s Disease
|Metabolites|2017|158
Alzheimer's disease–like pathology has transient effects on the brain and blood metabolome
|Neurobiology of Aging|2015|129
A novel dual GLP-1 and GIP receptor agonist is neuroprotective in the MPTP mouse model of Parkinson′s disease by increasing expression of BNDF
|Brain Research|2015|98