The E3 Ubiquitin Ligase SCF Cyclin F Promotes Sequestosome-1/p62 Insolubility and Foci Formation and is Dysregulated in ALS and FTD Pathogenesis
Jennilee M. Davidson(Macquarie University), Albert Lee(Macquarie University), Roger S. Chung(Macquarie University), Marco Morsch(Macquarie University), Stephanie L. Rayner(Macquarie University), Rachelle Balez(Illawarra Health and Medical Research Institute), Carlo Russo(Université Paris Cité), Kelly L. Williams(Biogen (United States)), Antonio Di Ieva(Macquarie University), Ian P. Blair(Macquarie University), Lezanne Ooi(University of Wollongong), Flora Cheng(Macquarie University), Kimberley Duncan(Macquarie University), Natalie M. Scherer(Macquarie University), Michelle Newbery(University of Wollongong), Sharlynn Wu(Macquarie University), Alberto Rábano(Fundacion Centro De Investigacion De Enfermedades Neurologicas), Lívia Rosa-Fernandes(Motor Neurone Disease Association), Kunjie Ding(Macquarie University), Alberto García‐Redondo, Shu Yang(Macquarie University)
Cited by 15
Related Papers
Exome sequencing in amyotrophic lateral sclerosis identifies risk genes and pathways
|Science|2015|980
TRPV3 is a temperature-sensitive vanilloid receptor-like protein
|Nature|2002|851
Transcription factor NFE2L2/NRF2 is a regulator of macroautophagy genes
|Autophagy|2016|409
Chromatin crosstalk in development and disease: lessons from REST
|Nature Reviews Genetics|2007|404
Neuronal hyperexcitability in Alzheimer’s disease: what are the drivers behind this aberrant phenotype?
|Translational Psychiatry|2022|392