SNAP25 disease mutations change the energy landscape for synaptic exocytosis due to aberrant SNARE interactions
Anna Kádková(University of Copenhagen), Jakob B. Sørensen(University of Copenhagen), Jacqueline Murach(Heidelberg University), Fabio Lolicato(University of Helsinki), Andrea Malsam(Heidelberg University), Maiken Østergaard(University of Copenhagen), Thomas H. Söllner(Heidelberg University), Jörg Malsam(Heidelberg University), Walter Nickel(Heidelberg University)
Cited by 11
Related Papers
Differential Control of the Releasable Vesicle Pools by SNAP-25 Splice Variants and SNAP-23
|Cell|2003|369
Sequential N‐ to C‐terminal SNARE complex assembly drives priming and fusion of secretory vesicles
|The EMBO Journal|2006|310
Loss of ceramide synthase 3 causes lethal skin barrier disruption
|Human Molecular Genetics|2011|275