The SWI/SNF chromatin-remodeling subunit DPF2 facilitates NRF2-dependent antiinflammatory and antioxidant gene expression
Glòria Mas Martín(University of Miami), Stephen D. Nimer(University of Miami), Francisco Vega(The University of Texas MD Anderson Cancer Center), Daniel Bilbao(University of Miami), Adnan K. Mookhtiar(Sylvester Comprehensive Cancer Center), Lluís Morey(University of Miami), Kranthi Kunkalla(Sylvester Comprehensive Cancer Center), Clayton K. Collings(Broad Institute), Scott C. Kogan(University of California, San Francisco), Francesco Tamiro(Sylvester Comprehensive Cancer Center), Felipe Beckedorff(Sylvester Comprehensive Cancer Center), Cigall Kadoch(Broad Institute), Stéphanie Duffort(Sylvester Comprehensive Cancer Center), Alfredo M. Valencia(Broad Institute), Daniel Karl(Sylvester Comprehensive Cancer Center), Ramin Shiekhattar(University of Miami), Concepcion Martinez-Caja(Sylvester Comprehensive Cancer Center), Chuan Chen(Tianjin University of Traditional Chinese Medicine), Na Man(ShanghaiTech University), Yuichiro Nakata(University of Miami), Hidehiro Itonaga(Nagasaki University)
Cited by 19
Related Papers
Activating RNAs associate with Mediator to enhance chromatin architecture and transcription
|Nature|2013|858
Epigenetic mechanisms in breast cancer therapy and resistance
|Nature Communications|2021|457
A non-canonical SWI/SNF complex is a synthetic lethal target in cancers driven by BAF complex perturbation
|Nature Cell Biology|2018|435