Tissue-resident, extravascular Ly6c− monocytes are critical for inflammation in the synovium
Anna B. Montgomery(Northwestern University), Shang Yang Chen(Northwestern University), Yidan Wang(Northwestern University), Gaurav Gadhvi(Northwestern University), Maximilian G. Mayr(Northwestern University), Carla M. Cuda(Northwestern University), Salina Dominguez(Northwestern University), Hadijat-Kubura M. Makinde(Northwestern University), Miranda G. Gurra(Northwestern University), Alexander V. Misharin(Northwestern University), Arthur M. Mandelin(Northwestern University), Eric Ruderman(Northwestern University), Anjali Thakrar(Northwestern University), Simran Brar(Northwestern University), Mary Carns(Northwestern University), Kathleen Aren(Northwestern University), Mahzad Akbarpour(Northwestern University), Andrew Filer(University Hospitals Birmingham NHS Foundation Trust), Saba Nayar(University Hospitals Birmingham NHS Foundation Trust), Ana Teososio(University of Birmingham), Triin Major(University of Birmingham), Ankit Bharat(Northwestern University), G. R. Scott Budinger(Northwestern University), Deborah R. Winter(Northwestern University), Harris Perlman(Northwestern University)
Cited by 12Open Access
Abstract
TR-MCs. These findings show a facet of mononuclear cell biology that could be imperative to understanding tissue-resident myeloid cell function in RA.
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