Cofilin activation in pancreatic acinar cells plays a pivotal convergent role for mediating CCK-stimulated enzyme secretion and growth

Irene Ramos-Álvarez(National Institutes of Health), Robert T. Jensen(National Institute of Diabetes and Digestive and Kidney Diseases)

Frontiers in Physiology

April 17, 2023

10.3389/fphys.2023.1147572

Cited by 4

Related Papers

Pituitary Adenylate Cyclase-Activating Polypeptide Causes Tyrosine Phosphorylation of the Epidermal Growth Factor Receptor in Lung Cancer Cells

|Journal of Pharmacology and Experimental Therapeutics|2012|43

Comparative Pharmacology of Bombesin Receptor Subtype-3, Nonpeptide Agonist MK-5046, a Universal Peptide Agonist, and Peptide Antagonist Bantag-1 for Human Bombesin Receptors

|Journal of Pharmacology and Experimental Therapeutics|2013|35

Gastrointestinal Hormones Cause Rapid c-Met Receptor Down-regulation by a Novel Mechanism Involving Clathrin-mediated Endocytosis and a Lysosome-dependent Mechanism

|Journal of Biological Chemistry|2006|15

Gastrin-Releasing Peptide Receptors Stimulate MAPK-Mediated Growth of Lung Cancer Cells by Transactivating HER4 in a Neuregulin-1, MAP Kinase-Dependent Manner Requiring Activation of the ROS-System

|Biology|2025|2

Bombesin Receptor Subtype-3 Regulates Tumor Growth by HER2 Tyrosine Phosphorylation in a Reactive Oxygen Species-Dependent Manner in Lung Cancer Cells

|Targets|2025|0