Age‐dependent loss of hepatic <scp>SIRT1</scp> enhances NLRP3 inflammasome signaling and impairs capacity for liver fibrosis resolution
Jennifer Adjei‐Mosi(The University of Texas at San Antonio Health Science Center), Mengwei Zang(Boston University), Nicolas Musi(Audie L. Murphy Memorial VA Hospital), Zerong Liang(Red Cross Hospital), Masahiro Morita(The University of Texas at San Antonio Health Science Center), Gavyn Lee Shealy(The University of Texas at San Antonio Health Science Center), Steven Blake Smithson(The University of Texas at San Antonio Health Science Center), Krupa Dhruvitha Amerineni(The University of Texas at San Antonio Health Science Center), Mei Wang(Wuhan University), Qing Sun(The University of Texas at San Antonio Health Science Center), Hanqing Chen(Capital Medical University), Jingyan Han(Boston University), Amrita Kamat(Longevity Biotech (United States)), Qinggong Ping(The University of Texas at San Antonio Health Science Center)
Cited by 48
Related Papers
Senolytics in idiopathic pulmonary fibrosis: Results from a first-in-human, open-label, pilot study
|EBioMedicine|2019|1.2k
SIRT1 Regulates Hepatocyte Lipid Metabolism through Activating AMP-activated Protein Kinase
|Journal of Biological Chemistry|2008|778
Polyphenols Stimulate AMP-Activated Protein Kinase, Lower Lipids, and Inhibit Accelerated Atherosclerosis in Diabetic LDL Receptor–Deficient Mice
|Diabetes|2006|654
AMP-activated Protein Kinase Is Required for the Lipid-lowering Effect of Metformin in Insulin-resistant Human HepG2 Cells
|Journal of Biological Chemistry|2004|454