Pathological mechanisms of cigarette smoking, dietary, and sedentary lifestyle risks in vascular dysfunction: mitochondria as a common target of risk factors
Sergey Dikalov(Vanderbilt University Medical Center), Anna Dikalova(Vanderbilt University Medical Center)
Cited by 12
Related Papers
DC isoketal-modified proteins activate T cells and promote hypertension
|Journal of Clinical Investigation|2014|533
Uncoupled Cardiac Nitric Oxide Synthase Mediates Diastolic Dysfunction
|Circulation|2010|254
Tobacco smoking induces cardiovascular mitochondrial oxidative stress, promotes endothelial dysfunction, and enhances hypertension
|American Journal of Physiology-Heart and Circulatory Physiology|2019|208