Genetic modifiers modulate phenotypic expression of tafazzin deficiency in a mouse model of Barth syndrome
Suya Wang(Huazhong Agricultural University), William T. Pu(Brigham and Women's Hospital)
Cited by 23
Related Papers
Endothelial-to-mesenchymal transition contributes to cardiac fibrosis
|Nature Medicine|2007|2.2k
Modeling the mitochondrial cardiomyopathy of Barth syndrome with induced pluripotent stem cell and heart-on-chip technologies
|Nature Medicine|2014|843
Single-Cell Resolution of Temporal Gene Expression during Heart Development
|Developmental Cell|2016|441
MicroRNA-1 Negatively Regulates Expression of the Hypertrophy-Associated Calmodulin and Mef2a Genes
|Molecular and Cellular Biology|2009|394
Co-occupancy by multiple cardiac transcription factors identifies transcriptional enhancers active in heart
|Proceedings of the National Academy of Sciences|2011|370