Release of hepatic xanthine oxidase (XO) to the circulation is protective in intravascular hemolytic crisis
Heidi M. Schmidt(University of Pittsburgh), Adam C. Straub(University of Pittsburgh), Julia Xu(National Institutes of Health), Katherine C. Wood(University of Pittsburgh), Madison Seman(West Virginia University), Sara E. Lewis(West Virginia University), Gowtham K. Annarapu(University of Pittsburgh), Mara Carreño(University of Pittsburgh), Werner J. Geldenhuys(Texas Tech University Health Sciences Center), Evan DeVallance(West Virginia University), Eric E. Kelley(West Virginia University), Sruti Shiva(University of Pittsburgh), Darío A. Vitturi, Emily A. Hileman(West Virginia University), Scott Hahn(University of Pittsburgh), Brooke A. Maxwell(West Virginia University)
Cited by 14
Related Papers
PINK1 deficiency impairs mitochondrial homeostasis and promotes lung fibrosis
|Journal of Clinical Investigation|2014|613
Inhaled NO accelerates restoration of liver function in adults following orthotopic liver transplantation
|Journal of Clinical Investigation|2007|227
Chemoptogenetic damage to mitochondria causes rapid telomere dysfunction
|Proceedings of the National Academy of Sciences|2019|148
AltitudeOmics: Red Blood Cell Metabolic Adaptation to High Altitude Hypoxia
|Journal of Proteome Research|2016|131
Fatty acid nitroalkenes ameliorate glucose intolerance and pulmonary hypertension in high-fat diet-induced obesity
|Cardiovascular Research|2014|95