TP53-mediated clonal hematopoiesis confers increased risk for incident atherosclerotic disease
Seyedeh M. Zekavat(Broad Institute), Derek Klarin(Broad Institute), Alexander G. Bick(Vanderbilt University), Nuria Matesanz(Spanish National Centre for Cardiovascular Research), James P. Pirruccello(Broad Institute), Alba Ferrer-Pérez(Spanish National Centre for Cardiovascular Research), Mark Trinder(Broad Institute), Marta Amorós-Pérez(MACOM (United States)), Vanesa Viana‐Huete(Spanish National Centre for Cardiovascular Research), Abhishek Niroula(Broad Institute), Peter Libby(Harvard University), Pradeep Natarajan(Broad Institute), Christopher J. Gibson(Otsuka (Japan)), Christie M. Ballantyne(Manpower Demonstration Research Corporation), Alejandra Aroca-Crevillén(Spanish National Centre for Cardiovascular Research), Kaavya Paruchuri(Broad Institute), Roberta Carriero(IRCCS Humanitas Research Hospital), Marzyeh Ghassemi(Massachusetts Institute of Technology), Benjamin L. Ebert(Broad Institute), Valentı́n Fuster(Spanish National Centre for Cardiovascular Research), Hongyu Zhao(Yale University), Saman Doroodgar Jorshery(Vector Institute), Scott M. Damrauer(Cardiovascular Institute of the South), María A. Zuriaga(Boston University), Andrés Hidalgo(Instituto de Salud Carlos III), José J. Fuster(Spanish National Centre for Cardiovascular Research), Carolina M. Greco(Humanitas University), Paolo Kunderfranco(IRCCS Humanitas Research Hospital), Virginia Zorita(Spanish National Centre for Cardiovascular Research), Gabriel K. Griffin(Brigham and Women's Hospital), Md Mesbah Uddin(Broad Institute)
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