Essential role of the mitochondrial Na+/Ca2+ exchanger NCLX in mediating PDE2-dependent neuronal survival and learning
Maya Rozenfeld(Ben-Gurion University of the Negev), Israel Sekler(Ben-Gurion University of the Negev), Ivana Savic Azoulay(Ben-Gurion University of the Negev), Moran Melamed(Ben-Gurion University of the Negev), Michal Hershfinkel(Ben-Gurion University of the Negev), Tsipi Ben Kasus Nissim(Ben-Gurion University of the Negev), Ora Kofman(Ben-Gurion University of the Negev), Grace E. Stutzmann(Rosalind Franklin University of Medicine and Science), Alexandra Stavsky(Ben-Gurion University of the Negev)
Cited by 17
Related Papers
NCLX is an essential component of mitochondrial Na <sup>+</sup> /Ca <sup>2+</sup> exchange
|Proceedings of the National Academy of Sciences|2009|776
Zinc in the physiology and pathology of the CNS
|Nature reviews. Neuroscience|2009|759
A zinc-sensing receptor triggers the release of intracellular Ca <sup>2+</sup> and regulates ion transport
|Proceedings of the National Academy of Sciences|2001|257
Synaptically Released Zinc Triggers Metabotropic Signaling via a Zinc-Sensing Receptor in the Hippocampus
|Journal of Neuroscience|2009|229
Role of GPR40 in fatty acid action on the β cell line INS-1E
|Biochemical and Biophysical Research Communications|2005|222