Universal clinical Parkinson’s disease axes identify a major influence of neuroinflammation
Cynthia Sandor(Broad Institute), Caleb Webber(University of Oxford), Yoav Ben‐Shlomo(University of Bristol), Jonathan Marchini(University of Oxford), Ann‐Kathrin Schalkamp(University of California, San Francisco), Leon Hubbard(Cardiff University), Andrew Dahl(University of Chicago), Donald G. Grosset(Queen Elizabeth University Hospital), Stephanie Millin(University of Oxford), Michael Lawton(University of Bristol), Nigel Williams(Cardiff University), Nabila Rahman(UK Dementia Research Institute)
Cited by 18
Related Papers
Common schizophrenia alleles are enriched in mutation-intolerant genes and in regions under strong background selection
|Nature Genetics|2018|1.7k
World Health Organization cardiovascular disease risk charts: revised models to estimate risk in 21 global regions
|The Lancet Global Health|2019|1.2k
Five amino acids in three HLA proteins explain most of the association between MHC and seropositive rheumatoid arthritis
|Nature Genetics|2012|960
The epidemiology of Parkinson's disease
|The Lancet|2024|809
High-density genetic mapping identifies new susceptibility loci for rheumatoid arthritis
|Nature Genetics|2012|677