Opposing Roles of IGFBP-3 and Heparanase in Regulating A549 Lung Cancer Cell SurvivalHind Al Khashali(Eastern Michigan University), Hedeel Guy Evans(Eastern Michigan University)CellsNovember 8, 202210.3390/cells11223533Cited by 9SaveCiteExport RISWatch citationsRelated PapersPhosphorylation of IGFBP-3 by Casein Kinase 2 Blocks Its Interaction with Hyaluronan, Enabling HA-CD44 Signaling Leading to Increased NSCLC Cell Survival and Cisplatin Resistance|Cells|2023|16Regulation of Vascular Endothelial Growth Factor Signaling by Nicotine in a Manner Dependent on Acetylcholine-and/or β-Adrenergic-Receptors in Human Lung Cancer Cells|Cancers|2023|8Regulation of Soluble E-Cadherin Signaling in Non-Small-Cell Lung Cancer Cells by Nicotine, BDNF, and β-Adrenergic Receptor Ligands|Biomedicines|2023|7Amyloid Beta Leads to Decreased Acetylcholine Levels and Non-Small Cell Lung Cancer Cell Survival via a Mechanism That Involves p38 Mitogen-Activated Protein Kinase and Protein Kinase C in a p53-Dependent and -Independent Manner|International Journal of Molecular Sciences|2024|6