Aggregatibacter actinomycetemcomitans Cytolethal Distending Toxin-Induces Cell Cycle Arrest in a Glycogen Synthase Kinase (GSK)-3-Dependent Manner in Oral Keratinocytes
Bruce J. Shenker(University of Pennsylvania), Kathleen Boesze‐Battaglia(University of Pennsylvania)
Cited by 14
Related Papers
Blockade of the PI-3K signalling pathway by the<i>A</i><i>ggregatibacter actinomycetemcomitans</i>cytolethal distending toxin induces macrophages to synthesize and secrete pro-inflammatory cytokines
|Cellular Microbiology|2014|55
Internalization of the Active Subunit of the Aggregatibacter actinomycetemcomitans Cytolethal Distending Toxin Is Dependent upon Cellugyrin (Synaptogyrin 2), a Host Cell Non-Neuronal Paralog of the Synaptic Vesicle Protein, Synaptogyrin 1
|Frontiers in Cellular and Infection Microbiology|2017|17
Peroxisome turnover and diurnal modulation of antioxidant activity in retinal pigment epithelia utilizes microtubule-associated protein 1 light chain 3B (LC3B)
|American Journal of Physiology-Cell Physiology|2019|16
Microsomal triglyceride transfer protein is necessary to maintain lipid homeostasis and retinal function
|The FASEB Journal|2024|13
Aggregatibacter actinomycetemcomitans cytolethal distending toxin modulates host phagocytic function
|Frontiers in Cellular and Infection Microbiology|2023|10