Inhibition of post-translational N-glycosylation by HRD1 that controls the fate of ABCG5/8 transporter
Douglas Cyr(University of North Carolina at Chapel Hill), Tappei Takada(University of Tokyo Hospital), Takashi Sato(Gunma University), Hirofumi Kai(Kumamoto University), Mary Ann Suico(Kumamoto University), Tsuyoshi Shuto(Kumamoto University), Hiroshi Suzuki(Niigata University), Masayuki Kaneko, Shingo Suzuki
Cited by 2
Related Papers
Decreased extra-renal urate excretion is a common cause of hyperuricemia
|Nature Communications|2012|703
Common Defects of ABCG2, a High-Capacity Urate Exporter, Cause Gout: A Function-Based Genetic Analysis in a Japanese Population
|Science Translational Medicine|2009|438
Gene Expression Profiling of Cells, Tissues, and Developmental Stages of the Nematode C. elegans
|Cold Spring Harbor Symposia on Quantitative Biology|2003|319
TDAG8 Is a Proton-sensing and Psychosine-sensitive G-protein-coupled Receptor
|Journal of Biological Chemistry|2004|262