An LKB1–mitochondria axis controls TH17 effector function
Francesc Baixauli(Department of Biomedicine Basel), George Caputa(Max Planck Institute of Immunobiology and Epigenetics), Gerhard Mittler(Max Planck Institute of Immunobiology and Epigenetics), Mario Fabri(University of California, Los Angeles), Hiromi Sesaki(Johns Hopkins University), Beth Kelly(Bloomberg (United States)), Cameron S. Field(Max Planck Institute of Immunobiology and Epigenetics), Klara Piletič(University of Oxford), Thomas Jenuwein(Max Planck Institute of Immunobiology and Epigenetics), Daniel J. Puleston(Bloomberg (United States)), David O’Sullivan(Max Planck Institute of Immunobiology and Epigenetics), Michal A. Stanczak(Bloomberg (United States)), David E. Sanin(Agency for Science, Technology and Research), Mai Matsushita(ETH Zurich), Erika L. Pearce(Bloomberg (United States)), Matteo Villa(Medical University of Graz), Katarzyna Duda(University of Copenhagen), Andrea Quintana(Max Planck Institute of Immunobiology and Epigenetics), Agnieszka M. Kabat(Bloomberg (United States)), Yaarub Musa(Max Planck Institute of Immunobiology and Epigenetics), Joerg M. Buescher(VIB-KU Leuven Center for Microbiology), Nisha Rana(Max Planck Institute of Immunobiology and Epigenetics), Katarzyna M. Grzes(Max Planck Institute of Immunobiology and Epigenetics), Lea J. Flachsmann(University of Freiburg), Mauro Corrado(TH Köln - University of Applied Sciences), Edward J. Pearce(Bloomberg (United States)), Joy Edwards-Hicks(University of Basel)
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