On the therapeutic potential of MAPK4 in triple-negative breast cancer
Fadia Boudghene-Stambouli(Institute for Research in Immunology and Cancer), Sylvain Meloche(Institute for Research in Immunology and Cancer)
Cited by 1
Related Papers
Activation Loop Phosphorylation of ERK3/ERK4 by Group I p21-activated Kinases (PAKs) Defines a Novel PAK-ERK3/4-MAPK-activated Protein Kinase 5 Signaling Pathway
|Journal of Biological Chemistry|2010|81
Activation of MEK1 or MEK2 isoform is sufficient to fully transform intestinal epithelial cells and induce the formation of metastatic tumors
|BMC Cancer|2008|68
Negative Regulation of MAPKK by Phosphorylation of a Conserved Serine Residue Equivalent to Ser212 of MEK1
|Journal of Biological Chemistry|2003|43
ERK3-MK5 signaling regulates myogenic differentiation and muscle regeneration by promoting FoxO3 degradation
|bioRxiv (Cold Spring Harbor Laboratory)|2021|2
Redundant roles of YES and SRC tyrosine kinases in driving malignant peripheral nerve sheath tumors
|bioRxiv (Cold Spring Harbor Laboratory)|2026|0