FPR2 contributes to the dysfunction of inflammation resolution and neuronal excitability in focal cortical dysplasia type IIb and tuberous sclerosis complexKaixuan Huang(Army Medical University), Shiyong Liu(Army Medical University)Research SquareJuly 21, 202210.21203/rs.3.rs-1843432/v1Cited by 0SaveCiteExport RISWatch citationsRelated PapersDownregulated FPR2 expression in the epileptogenic foci of focal cortical dysplasia type IIb and tuberous sclerosis complex patients|Research Square|2022|0